Chronic renal failure (cell therapy) lead to respiratory complications

Nausea and vomiting in patients with chronic renal failure is digestive dysfunction performance. Because each patient varying severity, and thus the number and degree of nausea and vomiting is different, but many of the clinical patients often complained to the doctor, morning nausea or vomiting increased, which is why it? First, we should understand due to renal function failure, blood urea nitrogen increased intestinal bacterial urease will break down urea into ammonia and stimulate the gastrointestinal mucosa and cause nausea, vomiting, nausea and vomiting and therefore an increased emphasis should and blood urea nitrogen. Renal failure, urinary dysfunction concentration, nocturia lot of patients because of massive loss of moisture at night, blood concentration, and thus the value of the relative increase in blood urea nitrogen morning, so early morning nausea and vomiting worse.

Chronic renal failure patients because of low internal environment disorders and immune function, and the occurrence of lung disease susceptible to in vivo pathogenicity factors, mainly uremia, pulmonary edema, pleural effusion and so on. Chronic renal failure

(1) uremic lung: also known as uremic pulmonary edema, pneumonia, uremia. Its symptoms are mild, only the early symptoms of uremia caused by the progression of the disease with the gradual emergence of mild to moderate cough, phlegm, and a small amount of breathing difficulties. When the development of interstitial fibrosis, dyspnea, and cyanosis increase. Small quantity is also an important symptom of hemoptysis. Need and cardiogenic pulmonary edema, pulmonary infection and pulmonary hemorrhage nephritic syndrome  differentiated.

(2) uremic pleural disease: incidence of 15% to 20%, and pleural friction rub, chest pain or chest discomfort, shortness of breath or fever. Pleural friction rub for 1 to 15 days, may be associated with bleeding. No relationship between blood urea nitrogen and bleeding.

(3) lung calcification: CRF often cause soft tissue calcification, lung is the most common site. The clinical manifestations of chronic dyspnea or acute, sub-acute respiratory failure, chest X-ray may be completely normal. Stop calcium, parathyroid resection, low phosphorus diet, oral application of alumina and low calcium dialysate to increase the number or duration of dialysis can reverse calcification.

(4) uremic pulmonary edema: is one of the common acute kidney Section. When CRF patients, especially oliguria, when no urine, such as sudden severe dyspnea, orthopnea, with fear, choking feeling, looking schungite, lips cyanosis, sweating, cough, expectoration, may be associated with hemoptysis, and a lot of pink frothy sputum, lungs full of symmetry crackles and wheezing, heart rate, pulse weak and should be considered acute pulmonary edema.

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